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Alcoholic hepatitis Reference – Symptoms, Diagnosis, Treatments
Diseases & Treatments Section @ BillDoll.com – The Billion Dollar Site
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Alcoholic hepatitis Reference – Symptoms, Diagnoses, Treatments
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Alcoholic liver disease-From Wikipedia, the free encyclopedia
Alcoholic liver disease Classification & external resources Microscopy of liver showing fatty change, cell necrosis, Mallory bodies ICD-10 K70 ICD-9 571.1 Alcoholic liver disease is the major cause of liver disease in Western countries, (in Asian countries, viral hepatitis is the major cause). It arises from the excessive ingestion of alcohol.
Contents 1 Pathophyisiology 1.1 Fatty change (K70.0) 1.2 Alcoholic hepatitis (K70.1) 1.3 Liver fibrosis (K70.2) 1.4 Alcoholic cirrhosis of liver (K70.3) and Alcoholic hepatic failure (K70.4) 2 External page 3 References
Pathophyisiology ICD-10 codes are provided below.
Fatty change (K70.0) Fatty change, or steatosis is the accumulation of fat in liver cells which can be seen as fatty globules under the microscope. Alcoholism causes large fatty globules (macrovesicular steatosis). Small fatty globules have different causes. Other causes of macrovesicular steatosis include diabetes, obesity and starvation. Alcoholic fatty change is probably dose related.
Alcoholic hepatitis (K70.1) Some people get an acute hepatitis or inflammatory reaction to the cells affected by fatty change. This is not directly related to the dose of alcohol. Some people seem more prone to this reaction than others. This is called alcoholic steatonecrosis and the inflammation probably predisposes to liver fibrosis.
Some signs and pathological changes in liver histology include:
Alcoholic hepatitis Mallory's Hyaline - a condition where pre-keratin filaments accumulate in hepatocytes. This sign is not limited to alcoholic liver disease, but is often characteristic.[1] Ballooning degeneration - hepatocytes in the setting of alcoholic change often swell up with excess fat, water and protein; normally these proteins are exported into the bloodstream. Accompanied with ballooning, there is necrotic damage. The swelling is capable of blocking nearly biliary ducts, leading to diffuse cholestasis.[1] Inflammation - Neutrophilic invasion is triggered by the necrotic changes and presence of cellular debris within the lobules. Ordinarily the amount of debris is removed by Kupffer cells, although in the setting of inflammation they become overloaded, allowing other white cells to spill into the parenchyma. These cells are particularly attracted to hepatocytes with Mallory bodies.[1] fibrosis - Cirrhosis - a progressive and permanent type of fibrotic degeneration of liver tissue.
Liver fibrosis (K70.2) Liver fibrosis, in itself, is largely asymptomatic but as it progresses it can turn into cirrhosis, where the fibrosis alters the architecture and impairs the function of the liver.
Fatty change and alcoholic hepatitis are probably reversible. The later stages of fibrosis and cirrhosis tend to be irreversible but can usually be quite well managed for long periods of time.
Alcoholic cirrhosis of liver (K70.3) and Alcoholic hepatic failure (K70.4) Cirrhosis is a late stage of liver disease marked by fibrosis and altered liver architecture. It is often progressive and may eventually lead to liver failure. Late complications of cirrhosis or liver failure include portal hypertension, coagulation disorders, ascites and other complications including hepatic encephalopathy and the hepatorenal syndrome.
Cirrhosis also has number of other causes, such as hepatitis and toxins. The late stages of cirrhosis (say from viral hepatitis or alcohol) may look similar. This phenomenon is termed a "final common pathway" for a disease. References ^ a b c Cotran; Kumar, Collins. Robbins Pathologic Basis of Disease. Philadelphia: W.B Saunders Company. 0-7216-7335-X. Retrieved from "http://en.wikipedia.org/wiki/Alcoholic_liver_disease"
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